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After five months, the researchers compared each set of mice to a control group that consumed the same diet but did not eicosapentaenoic acid carry the Alzheimer's genes. The results surprised them. "We found high amounts of synaptic damage in the brains of the Alzheimer's-diseased mice that ate the DHA-depleted diet," Frautschy said. "These changes closely resembled those we see in eicosapentaenoic acid the brains of people with Alzheimer's disease." Although the mice on the DHA-supplemented diet also carried the Alzheimer's genes, they still performed much better in memory testing than the mice in the eicosapentaenoic acid first group. "After adjusting for all possible variables, DHA was the only factor remaining that protected the mice against the synaptic damage and memory loss that should have resulted from their Alzheimer's genes," Cole said. "We concluded that the DHA-enriched diet was holding their genetic disease at bay." Increased DHA intake may stave off Alzheimer's. The present results provide, for the first time, evidence that the combination of genetic (mutant human APP) and environmental risk factors (dietary essential fatty acids) for AD can act synergistically to quantitatively reduce synaptic proteins, specifically, dendritic scaffold proteins, that are critical for cognition as evidenced by memory deficits observed in the Morris water
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