Vol. 166, No. 10 dry eye coconut fatty acid

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business news, hispanic research, glaucoma, female, coconut fatty acid , painful joints, masturbate, record types: paper, mercado hispano, hispanic census, ahrq, plump breasts , Some mice received food without DHA for 103 days and subsequently showed depleted stores of DHA in their brains. In contrast, mice getting chow supplemented with DHA had high brain concentrations of the fatty acid. Notably, these animals maintained good concentrations of two proteins that enable synapses to dry eye function, but dry eye DHA-deficient mice had insufficient concentrations of those substances. Commenting on the study, nutritionist Julie A. Conquer of the University of Guelph in Ontario says dry eye that these results show that DHA in the diet can affect the biochemistry of brain-signaling pathways. Cole and his team also tested memory in elderly mice that had received food with or without DHA for nearly 5 months. They trained the 21-month-old mice to swim to a platform in a tank of warm water, then raised the water level to submerge the platform slightly.
Vol. 166, No. 10 , p. 148 Alzheimer's Advance: Omega-3 fatty acid benefits mice Nathan Seppa A diet that includes a key omega-3 fatty acid found in fish and canola oil prevents some memory loss in mice that develop a disease similar to Alzheimer's, researchers report in the Sept. 2 Neuron. The finding is consistent with previous research coconut fatty acid suggesting that fish oil supplements might reduce the risk of Alzheimer's disease coconut fatty acid in people. Other coconut fatty acid work has shown that the fatty acid, called docosahexaenoic acid (DHA), is essential to brain function and that Alzheimer's patients have low concentrations of it in their blood. The early memory and learning problems that mark the disease occur because damaged brain cells fail to transmit messages consistently to each other across junctions called synapses. To assess what role DHA might have in maintaining this transmission, Greg M. Cole, a neuroscientist at the University of California, Los Angeles (UCLA) and his colleagues used old mice—17 months on average—that were genetically engineered to develop waxy protein plaques in their brains, much as Alzheimer's patients do.
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