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| breast feeding support, whole health, drug information, nurses' health study, food and mood, hair problems, tinnitus, ipo news, alpha–lipoic acid, queen fat bottom girls , ageing, irritable bowel syndrome, plump tgp , fishoil, n 3 polyunsaturated fatty acids , omaga 3 fatty acids, | In experimental animals and humans (students facing an raw milk academic examination), external stressors increase the production of inflammatory cytokines, raw milk such as IL-6, TNF and IFN [82], and serum PUFA levels predict the response of proinflammatory cytokines to psychologic stress [83]. The increased C20:46/C20:53 ratio and the imbalance in the omega-6/omega-3 PUFA ratio in major depression may be related to the increased production of proinflammatory cytokines and eicosanoids in that illness [79]. The increased omega-6/omega-3 ratio in Western diets most likely contributes to an increased incidence of cardiovascular disease and inflammatory raw milk disorders. Lower serum HDL cholesterol and an increased C20:46/C20:53 ratio are related both to depression and to a higher risk of cardiovascular disease, which shows a strong comorbidity with depression [79,80]. |
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| There is evidence that changes in fatty acid composition are involved in the pathophysiology of major depression. Changes in serotonin (5-HT) receptor number and function caused by changes in PUFA provide the theoretical rationale connecting fatty acids with the current receptor and neurotransmitter breast feeding support theories of depression [79–81]. The involvement of breast feeding support changes in fatty acid composition in the pathophysiology of major depression also revolves around its role in immune function and production of cytokines. There is now evidence that major depression is accompanied by an acute phase response, breast feeding support increased secretion of eicosanoids, such as prostaglandins; cytokines, i.e. the monocyte cytokines, IL-1ß and IL-6, as well as the Th-1-like cytokines, IL-2 and IFN. IL-1, IL-2 and TNF activate the hypothalamic adrenal (HPA) axis where proinflammatory cytokines can induce resistance to the effects of glucocorticoid hormones by influencing glucocorticoid receptor expression. |
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